Author: admin_alscvf8679

What Are The Benefits of Amyotrophic Lateral Sclerosis Support Groups?
20 Mar 2020admin_alscvf8679

Are you currently living with a person experiencing amyotrophic lateral sclerosis or ALS? It is best to find the right environment to cater, treat, and understand the needs of a person living with a rare disease. Amyotrophic lateral sclerosis support groups can give further and enhanced protocols and guidelines that may encourage those struggling in this motor movement disorder. Moreover, most ALS support groups and ALS association differ in age. Hence, patients, family members, and loved ones may have a better perspective on how to deal with this ongoing rare disease crisis. Check out other beneficial facts from your primary health care physician on what are the risks of amyotrophic lateral sclerosis from the nearest hospital.
Amyotrophic Lateral Sclerosis Drugs
20 Mar 2020admin_alscvf8679

Are there amyotrophic lateral sclerosis drugs? What are the drugs that can help patients cope with their amyotrophic lateral sclerosis? …
What Are The Risks Of Having Primary Lateral Sclerosis? (Basic Details)
20 Mar 2020admin_alscvf8679

The central nervous system (CNS) is probably one of the most if not the top sensitive parts of our body. Our mind and body functions simultaneously, thanks to our brain and nerves. However, any disruption, blockage, or destruction in these nerves may lead to problems that affect the whole movement of legs, arms, and voluntary muscles that a person have. ALS or Amyotrophic lateral sclerosis is similar to progressive primary lateral sclerosis as both can hinder neurons for motor movement.
What Are The Stages Of Amyotrophic Lateral Sclerosis?
20 Mar 2020admin_alscvf8679

What are the stages of amyotrophic lateral sclerosis? How severe is this disease in the first place? This article will talk about amyotrophic lateral sclerosis, the stages of this disease and other important facts about it.

A Cluster of Autism-Associated Variants on X-Linked NLGN4X Functionally Resemble NLGN4Y 04/02/2020

NLGN4X and NLGN4Y are thought to have similar functions due to their ∼97% shared amino acid identity. Nguyen et al. demonstrate NLGN4Y has a deficit in trafficking due to one amino acid difference. The trafficking deficit phenotype is also found in a cluster of autism-associated variants in NLGN4X near the critical amino acid.

Basolateral Amygdala Drives a GPCR-Mediated Striatal Memory Necessary for Predictive Learning to Influence Choice 04/01/2020

Predictive learning guides future choices between actions. Here, Morse et al. demonstrate that this fundamental decision-making process is mediated by a novel GPCR-based memory involving the expression of delta-opioid receptors on cholinergic interneurons in the nucleus accumbens shell.

All-Electrical Ca2+-Independent Signal Transduction Mediates Attractive Sodium Taste in Taste Buds 03/30/2020

Nomura et al. report the identity of taste cells dedicated to the Na+-selective, attractive component of salty taste (a.k.a. sodium taste) and their intracellular signal transduction. In taste cells expressing ENaC and CALHM1/3, ENaC-mediated Na+ influx generates suprathreshold depolarization for action potentials driving voltage-dependent neurotransmitter release via the CALHM1/3-channel synapse.

The Loss of TBK1 Kinase Activity in Motor Neurons or in All Cell Types Differentially Impacts ALS Disease Progression in SOD1 Mice 03/27/2020

DNA sequence variants in the TBK1 gene associate with familial and sporadic ALS. Gerbino et al. show that partial or complete loss-of-function TBK1 mutations alone do not induce neurodegeneration in mice. However, they profoundly affect disease onset and progression in the SOD1 ALS mouse model.